Law research found that a viral protein can be transformed into a cervical cancer "killer"

Researchers from the University of Louis Pasteur in Strasbourg, France, have made a groundbreaking discovery: a small mutation in a protein linked to cervical cancer can actually stop the disease from spreading. This finding could pave the way for new and more effective treatments for cervical cancer, offering hope to millions of women worldwide. According to a report published in the online edition of the British Journal of Oncogene on the 17th, approximately 500,000 women are diagnosed with cervical cancer each year, with 80% living in low-income countries. The primary cause of this disease is the human papillomavirus (HPV), which spreads mainly through sexual contact. The research team found that HPV produces two key proteins, E6 and E7. Normally, E6 promotes the growth of cancer cells, but when it undergoes a specific mutation, it starts to suppress tumor development instead. This tiny genetic change, though seemingly minor, completely alters the function of the E6 protein, turning it from a harmful agent into a potential therapeutic target. Currently, the main approach to preventing cervical cancer is vaccination. However, high costs limit access in many developing regions, and vaccines are not effective for women who are already infected. With this new discovery, scientists hope to develop targeted drugs that can treat the disease more effectively, especially for those who are already suffering from it. This breakthrough highlights the importance of understanding the molecular mechanisms behind cancer and opens up new possibilities for innovative therapies.

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